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HERO ID
4843574
Reference Type
Journal Article
Title
Zinc deficiency in molybdenum poisoned cattle
Author(s)
Parada, R
Year
1981
Is Peer Reviewed?
1
Journal
Veterinary and Human Toxicology
ISSN:
0145-6296
Volume
23
Issue
1
Page Numbers
16-21
Abstract
HEEP COPYRIGHT: BIOL ABS. Clinical signs ascribable to Zn deficiency were noted in Friesian cows industrially poisoned with Mo. Zn, Cu and Mo were determined in blood serum and black hair, and in the contaminated alfalfa pasture the group grazed on. Hematological parameters, and serum Ca and alkaline phosphatase activity, were also determined. Pooled samples of alfalfa from 2 uncontaminated pastures, and of blood, serum and black hair of clinically normal Friesian cattle grazing on these were used as controls. A mixed contamination of the polluted pasture with Mo and Cu was found, both metals being inversely correlated with the distance to the polluting chimney. Zn concentrations were normal and not significantly correlated with the distance to the chimney. Very high Mo was found in serum and hair of the poisoned animals; Cu was normal in serum and hair. Low Ca and alkaline phosphatase activity were found in serum, both variables significantly correlated with serum Zn. Reduced red blood cell number, packed cell volumes and Hb concentrations, but no significant correlation of these parameters with any of the trace metals in serum or hair, were found. Signs ascribed to Zn deficiency were consistent with the reduction of Zn in serum and hair and decreased alkaline phosphatase activity in serum. A Zn deficiency conditioned by a simultaneous increased intake of Mo and Cu is proposed.
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IRIS
•
Molybdenum
Litsearch 2018
Toxline
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