Glucocorticoid hormone effect on transplacental carcinogenesis and lung differentiation: influence of histocompatibility-2 complex

Oomen, LC; van der Valk, MA; Hart, AA; Demant, P

HERO ID

1419690

Reference Type

Journal Article

Year

1989

Language

English

PMID

2921775

HERO ID 1419690
In Press No
Year 1989
Title Glucocorticoid hormone effect on transplacental carcinogenesis and lung differentiation: influence of histocompatibility-2 complex
Authors Oomen, LC; van der Valk, MA; Hart, AA; Demant, P
Journal Journal of the National Cancer Institute
Volume 81
Issue 7
Page Numbers 512-517
Abstract In the mouse, the histocompatibility-2 (H-2) haplotype influences induction of lung and intestinal tumors by N-ethyl-N-nitrosourea (ENU) treatment of fetuses or infant mice. The differentiation of lung and intestinal epithelium is known to be regulated by glucocorticoids. We show that glucocorticoid-induced development of alveolar lung volume is H-2 influenced and that glucocorticoid treatment of fetuses also influences prenatal ENU induction of lung and intestinal tumors. These glucocorticoid effects on tumorigenesis are also H-2 influenced. The number of papillary lung tumors increased in B10 (H-2b) and decreased in B10.A (H-2a) mice. In the intestine, the number of tumors increased in H-2b females and decreased in H-2b males. In H-2a mice, the number of intestinal tumors was unchanged but their location was altered. We propose that the H-2 complex influences tumorigenesis in lung and small intestine by affecting the hormonal regulation of differentiation of target epithelial cells.
Pmid 2921775
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English