Regulation of TH2 responses by the pulmonary Clara cell secretory 10-kd protein

Hung, CH; Chen, LC; Zhang, Z; Chowdhury, B; Lee, WL; Plunkett, B; Chen, CH; Myers, AC; Huang, SK

HERO ID

1639191

Reference Type

Journal Article

Year

2004

Language

English

PMID

15356574

HERO ID 1639191
In Press No
Year 2004
Title Regulation of TH2 responses by the pulmonary Clara cell secretory 10-kd protein
Authors Hung, CH; Chen, LC; Zhang, Z; Chowdhury, B; Lee, WL; Plunkett, B; Chen, CH; Myers, AC; Huang, SK
Journal Journal of Allergy and Clinical Immunology
Volume 114
Issue 3
Page Numbers 664-670
Abstract <strong>BACKGROUND: </strong>Pulmonary Clara cell secretory 10-kd protein (CC10) is a steroid-inducible and potentially anti-inflammatory cytokine, but its direct involvement in the regulation of T-cell responses remains unknown.<br /><br /><strong>OBJECTIVE: </strong>The role of CC10 in the regulation of T(H)2 cytokine expression was investigated.<br /><br /><strong>METHODS: </strong>The levels of cytokine and GATA-3 expression were determined by ELISA and RT-PCR, respectively. Bronchoalveolar lavage fluid cell counts were also determined by using a standard protocol. CC10 expression in vivo was determined by immunocytochemistry and Western blotting.<br /><br /><strong>RESULTS: </strong>In vitro, a significant, dose-dependent suppressive effect of CC10 was found on T(H)2 cytokine expression, but not IFN-gamma, in splenocytes of antigen-sensitized mice. A similar suppressive effect was also noted in polarized CD4(+) T(H)2 cells, but not in naive CD4(+) T cells. In contrast, CC10 was able to induce IFN-gamma expression in naive CD4(+) T cells, but not in polarized T(H)1 cells. Furthermore, the suppression of T(H)2 cytokine expression was concomitant with reduction of a critical transcription factor, GATA-3. Of significance was the finding that although no significant change was found in the decay kinetics of T(H)2 cytokine transcripts, a significant decrease in mRNA stability of GATA-3 was seen in CC10-treated cells. In vivo, reconstitution of the CC10 gene in CC10-deficient mice resulted in significantly lower levels of T(H)2 cytokines, concomitant with a decrease in GATA-3 expression, after challenge with Ag compared with those seen in mock-transduced mice, which are associated with reduced levels of pulmonary eosinophilia.<br /><br /><strong>CONCLUSION: </strong>These results demonstrate, that CC10 plays a direct role in the regulation of T-cell-mediated inflammatory responses.
Doi 10.1016/j.jaci.2004.05.042
Pmid 15356574
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English