MiR-193a-3p promotes the multi-chemoresistance of bladder cancer by targeting the HOXC9 gene

Lv, L; Li, Y; Deng, H; Zhang, C; Pu, Y; Qian, L; Xiao, J; Zhao, W; Liu, Q; Zhang, D; Wang, Y; Zhang, H; He, Y; Zhu, J

HERO ID

2902356

Reference Type

Journal Article

Year

2015

Language

English

PMID

25444900

HERO ID 2902356
In Press No
Year 2015
Title MiR-193a-3p promotes the multi-chemoresistance of bladder cancer by targeting the HOXC9 gene
Authors Lv, L; Li, Y; Deng, H; Zhang, C; Pu, Y; Qian, L; Xiao, J; Zhao, W; Liu, Q; Zhang, D; Wang, Y; Zhang, H; He, Y; Zhu, J
Journal Cancer Letters
Volume 357
Issue 1
Page Numbers 105-113
Abstract Chemoresistance prevents the curative cancer chemotherapy and presents a formidable challenge for both cancer researchers and clinicians. We have previously shown that miR-193a-3p promotes the multi-chemoresistance of bladder cancer cells via repressing its three target genes: SRSF2, PLAU and HIC2. Here, we showed that as a new direct target, the homeobox C9 (HOXC9) gene also executes the promoting effect of miR-193a-3p on the bladder cancer chemoresistance from a systematic study of multi-chemosensitive (5637) and resistant (H-bc) bladder cancer cell lines in both cell culture and tumor-xenograft/nude mice system. Paralleled with the changes in the drug-triggered cell death, the activities of both DNA damage response and oxidative stress pathways were drastically altered by a forced reversal of miR-193a-3p or HOXC9 levels in bladder cancer cells. In addition to a new mechanistic insight, our results provide a set of the essential genes in the miR-193a-3p/HOXC9/DNA damage response/oxidative stress pathway axis as the diagnostic targets for the guided anti-bladder cancer chemotherapy.
Doi 10.1016/j.canlet.2014.11.002
Pmid 25444900
Wosid WOS:000348084100010
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English