The role of mouse intestinal microflora in the metabolism of trichloroethylene, an in vivo study

Moghaddam, AP; Abbas, R; Fisher, JW; Lipscomb, JC

HERO ID

630794

Reference Type

Journal Article

Year

1997

Language

English

PMID

9426363

HERO ID 630794
In Press No
Year 1997
Title The role of mouse intestinal microflora in the metabolism of trichloroethylene, an in vivo study
Authors Moghaddam, AP; Abbas, R; Fisher, JW; Lipscomb, JC
Journal Human & Experimental Toxicology
Volume 16
Issue 11
Page Numbers 629-635
Abstract 1. Both trichloroethylene and its metabolite, dichloroacetic acid, produce liver tumors peroxisome proliferation and other adverse cellular alterations in rodents. 2. The hepatic mechanism by which dichloroacetic acid is formed is not conclusively demonstrated, but pharmacokinetic models have successfully associated its formation with trichloroacetic acid as immediate precursor. 3. Previous investigations have shown that dichloroacetic acid is formed from trichloroacetic acid by gut microflora isolated in vitro. 4. To determine the impact of gut microflora on dichloroacetic acid formation from a trichloroethylene dose in vivo, we developed a procedure which reduced gut microflora some 3 orders of magnitude below published levels. 5. The administration of trichloroethylene to control mice and to mice whose gut was practically sterile resulted in equivalent concentrations of dichloroacetic acid and other metabolites in blood and liver, but significantly different content of these metabolites in cecum contents. 6. These data indicate that gut microflora contribute minimally, if at all, to the formation of circulating dichloroacetic acid under these conditions
Doi 10.1177/096032719701601101
Pmid 9426363
Is Certified Translation No
Dupe Override No
Is Public Yes
Language Text English
Keyword Air; Animals; Bacteria; blood; Cecum; Dichloroacetate; Intestines; Liver; Male; metabolism; Mice; microbiology; Ohio; Research; Solvents; Toxicology; Trichloroacetic Acid; Trichloroethylene
Is Qa No