Health & Environmental Research Online (HERO)


Print Feedback Export to File
604833 
Journal Article 
Streptozotocin ile Diyabet OluÅŸturulmuÅŸ Sıçanlarda Kafeik Asit Fenetil Ester (CAPE) Tedavisinin Retinadaki MDA, NO Düzeyleri ve Antioksidan Enzim Aktiviteleri Üzerine Etkileri 
Durmuş, M; Yilmaz, HR; Uz, E; Özçelik, N 
2008 
38 
525-530 
Aim: This study was performed to determine the effect of caffeic acid phenethyl ester (CAPE) treatment on levels of malondialdehyde (MDA) and nitric oxide (NO) and activities of antioxidant enzymes in retinas of streptozotocin (STZ)-induced diabetic rats. Materials and Methods: Twenty-seven rats were enrolled and divided into three groups: group 1: nondiabetic rats as control (n = 9); group 2: STZ-induced, untreated diabetic rats (n = 8); and group 3: STZ-induced, CAPE-treated diabetic rats (n = 10). Levels of MDA and NO and the activities of superoxide dismutase (SOD) and glutathione peroxidase (GSH-Px) in retinal tissues were analyzed. Results: In retinas of untreated diabetic rats, levels of MDA and NO and the activity of GSH-Px were significantly increased (P = 0.001, P = 0.0001, P = 0.0001, respectively), while the SOD activity was significantly decreased (P = 0.021) when compared to the control group. GSH-Px activity in the CAPE-treated diabetic group was markedly higher than that of the control group (P = 0.0001). MDA and NO levels were lower (P = 0.005, P = 0.001, respectively) and SOD activity was higher in the CAPE-treated diabetic group compared to the untreated diabetic group (P = 0.016). Conclusions: It can be concluded that nitrosative and oxidative stress is increased in the diabetic retina. CAPE treatment inhibits lipid peroxidation and NO overproduction and regulates SOD enzyme activities in diabetic rat retinas while it has no effects on GSH-Px activity. (English) [ABSTRACT FROM AUTHOR] Amaç: Bu çaliÅŸma, streptozotocin (STZ) ile diyabet oluÅŸturulmuÅŸ siçanlarda, kafeik asit fenetil ester (CAPE) tedavisinin retinadaki malondialdehit (MDA), nitrik oksit (NO) düzeyleri ve antioksidan enzim aktiviteleri üzerine etkilerini araÅŸtirmak üzere yapildi. Yöntem ve Gereç: Yirmiyedi siçan çaliÅŸmaya alindi ve üç gruba ayrildi: grup 1: diyabetik olmayan siçanlar (kontrol grubu) (N = 9); grup 2: STZ uygulanmiÅŸ-tedavi uygulanmamiÅŸ siçanlar (N = 8); grup 3: STZ uygulanmiÅŸ ve CAPE tedavisi verilmiÅŸ siçanlar (N = 10). Bütün gruplarin retinalarinda, MDA ve NO düzeyleri, superoksit dismutaz (SOD) ve glutatyon peroksidaz (GSH-Px) enzim aktiviteleri analiz edildi. Bulgular: Kontrol grubu ile kiyaslandiÄŸinda, tedavi uygulanmamiÅŸ diyabetik siçanlarda MDA ve NO düzeyleri ve GSH-Px aktivitesi artmiÅŸ ( sirasiyla P = 0.001, P = 0.0001, P = 0.0001), buna karÅŸilik SOD aktivitesi önemli ölçüde azalmiÅŸ olarak bulundu (P = 0.021). GSH-Px aktivitesi CAPE tedavisi uygulanmiÅŸ diyabetik siçanlarda, kontrol grubuna göre belirgin ölçüde yüksekti (P = 0.0001). CAPE tedavisi uygulanmiÅŸ diyabetik siçanlarda, tedavi edilmemiÅŸ olanlara nazaran MDA ve NO düzeyleri azalmiÅŸ (sirasiyla P = 0.005, P = 0.001), SOD aktivitesi artmiÅŸti (P = 0.016). Sonuç: STZ ile diyabet oluÅŸturulmuÅŸ siçanlarda MDA ve NO düzeyleri ve GSH-Px aktivitesi artmiÅŸti, CAPE tedavisi ile GSH-Px diÅŸindakiler azalmaktaydi. SOD aktivitesi diyabetik grupta azalmiÅŸken, CAPE tedavisi ile artmaktaydi. (Turkish) [ABSTRACT FROM AUTHOR] Copyright of Turkish Journal of Medical Sciences is the property of Scientific and Technical Research Council of Turkey and its content may not be copied or emailed to multiple sites or posted to a listserv without the copyright holder's express written permission. However, users may print, download, or email articles for individual use. This abstract may be abridged. No warranty is given about the accuracy of the copy. Users should refer to the original published version of the material for the full abstract. (Copyright applies to all Abstracts) 
MALONDIALDEHYDE; DIABETES; ANTIOXIDANTS; NITRIC oxide; SUPEROXIDE dismutase; TREATMENT; Antioxidant enzymes; caffeic acid phenethyl ester; diabetic retinopathy; lipid peroxidation; Antioksidan enzimler; diyabetik retinopati; kafeik asit fenetil ester; lipid peroksidasyonu