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7175274 
Journal Article 
Dual Targeting of CDK4/6 and BCL2 Pathways Augments Tumor Response in Estrogen Receptor-Positive Breast Cancer 
Whittle, , JR; Vaillant, F; Surgenor, E; Policheni, AN; Giner, G; Capaldo, BD; Chen, HR; Liu, HK; Dekkers, JF; Sachs, N; Clevers, H; Fellowes, A; Green, T; Xu, H; Fox, SB; Herold, MJ; Smyth, GK; Gray, DHD; Visvader, JE; Lindeman, GJ; , 
2020 
Clinical Cancer Research
ISSN: 1078-0432
EISSN: 1557-3265 
English 
Although cyclin-dependent kinase 4 and 6 (CDK4/6) inhibitors significantly extend tumor response in patients with metastatic estrogen receptor-positive (ER+) breast cancer, relapse is almost inevitable. This may, in part, reflect the failure of CDK4/6 inhibitors to induce apoptotic cell death. We therefore evaluated combination therapy with ABT-199 (venetoclax), a potent and selective BCL2 inhibitor.