Porphyromonas gingivalis-related cardiac cell apoptosis was majorly co-activated by p38 and extracellular signal-regulated kinase pathways | Health & Environmental Research Online (HERO)

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Citation
Tags

HERO ID

8624363

Reference Type

Journal Article

Title

Porphyromonas gingivalis-related cardiac cell apoptosis was majorly co-activated by p38 and extracellular signal-regulated kinase pathways

Author(s)

Lee, SD; Wu, CC; Kuo, WW; Lin, JA; Hwang, JM; Lu, MC; Chen, LM; Hsu, HH; Wang, CK; Chang, SH; Huang, CY; ,

Year

2006

Is Peer Reviewed?

Yes

Journal

Journal of Periodontal Research
ISSN: 0022-3484
EISSN: 1600-0765

Volume

Issue

Page Numbers

39-46

Language

English

PMID

16409254

DOI

10.1111/j.1600-0765.2005.00842.x

Web of Science Id

WOS:000234405000006

URL

http://://WOS:000234405000006
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Abstract

Little is known about the pathogenesis of apoptosis caused in cardiac tissues by periodontitis pathogens. The purpose of this study was to determine the related effect of periodontal pathogen Porphyromonas gingivalis on cardiac cell apoptosis.

Keywords

Apoptotic; Cardiac; Cell death; Perlodontltis; Porphyromonas gingivalis; Signal pathways