Health & Environmental Research Online (HERO)


Print Feedback Export to File
47157 
Journal Article 
Pathogenesis of hyperuricemia in saturnine gout 
Ball, GV; Sorensen, LB 
1969 
New England Journal of Medicine
ISSN: 0028-4793
EISSN: 1533-4406 
280 
22 
1199-1202 
English 
Thirty-seven of 43 admissions for gout to the Birmingham Veterans Administration Hospital in 1967 were for gout of the "saturnine" variety secondary to prolonged consumption of moonshine contaminated with lead. Uric acid kinetic studies done on three typical patients on a purine deprivation diet yielded similar results. Mean values were: uric acid turnover, 550 mg per day; uric acid pool, 1308 mg per day; urinary uric acid in percentage of turnover, 25.7; and uric acid recovered from urine for seven days, as percentage of dose, 24.9. Glycine-14C incorporation into urinary uric acid expressed as percentage of dose and corrected for extrarenal disposal was 0.102. These results confirm the role of the kidney in the pathogenesis of hyperuricemia in this disorder and the susceptibility of these patients to the development of gout.

*From the Division of Rheumatology, University of Alabama in Birmingham, the Medical Center and the Veterans Administration Hospital, Birmingham, and the Department of Medicine, University of Chicago, Chicago, 1ll. (address reprint requests to Dr. Ball at 1919 7th Ave., South, Birmingham, Ala. 35233).

Portions of this paper, delivered at a meeting of the American College of Physicians in Boston, Mass., April 1, 1967, have been published as an abstract entitled, "Pathogenesis of Saturnine Gout: Urate Kinetic Studies" in the Annals of Internal Medicine, Vol. 68, No. 5, May, 1968, p. 1178. 
NAAQS
• ISA-Lead (2013 Final Project Page)
     Considered
     Cited
          1st Draft
          2nd Draft
          3rd Draft
          Final
     Health Effects